Evidence of a Hereditary Factor Affecting the Chick’s Response to Uncomplicated Selenium Deficiency

摘要: Abstract Experiments were conducted to determine whether a hereditary factor accounted for part of the variation observed in growth responses young chicks uncomplicated selenium (Se) deficiency. Results showed that such influenced fed Se-deficient diet. Those most susceptible (SUS) depressing effects Se-deficiency oxidized14 CH3 -methionine 14CO2 at greater rate when diet than did appeared refractory (REF) (i.e., grew near normal rates) Se-deficiency. When diets supplemented with Se, both lines exhibited equivalent rates 14CH3-methionine oxidation. The SUS have lower levels free homocystine and cystathionine plasma compared REF or Se-supplemented lines. These results provide evidence an impairment sulfur-amino acid metabolism as basis effect.