作者: Alexander S. Chen , Renee D. Read
DOI: 10.1007/978-3-030-23629-8_12
关键词: Genetic screen 、 PI3K/AKT/mTOR pathway 、 Receptor tyrosine kinase 、 Biological pathway 、 Asymmetric cell division 、 Carcinogenesis 、 Gene 、 Drosophila melanogaster 、 Computational biology 、 Biology
摘要: … promotes GBM tumorigenesis remain poorly understood, ACAT1 … wherein ACAT1 expression was upregulated in human GBM cell lines in a EGFR-PI3K-dependent manner, and ACAT1 …