The striatonigral fibres and the feedback control of dopamine metabolism.
作者： I. F. Tulloch , G. W. Arbuthnott , A. K. Wright , M. Garcia-Munoz , N. M. Nicolaou
DOI: 10.1017/S0033291700016159
关键词:
摘要: It proved possible to make lesions which interrupted the striatonigral GABA-containing pathway in rat brain without causing concomitant damage nigrostriatal dopamine containing system. Estimations of striatal concentrations (DA), dihydroxyphenyl-acetic acid (DOPAC) and homovanillic (HVA) inducated that these had no influence either on normal DA turnover or enhancement induced by neuroleptics. Behavioural experiments suggested a motor output function for pathway.
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